Jaban M Moore alias Dr. Foot Fungus Fraud

Jaban M Moore is not licensed or approved by Missouri State Board of Chiropractic Examiners to advertise Toenail fungus that keeps coming back is almost always a sign of systemic Candida overgrowth, which thrives when the immune system is suppressed by mold exposure. as within their scope of practice.

Toenail fungus that keeps coming back is almost always a sign of systemic Candida overgrowth, which thrives when the immune system is suppressed by mold exposure.

Supports

There is some indirect support that Candida can infect nails in the setting of impaired immunity, and that recurrent nail Candida can be a manifestation of an underlying immunodeficiency. Reviews of onychomycosis note that yeasts such as Candida albicans and Candida parapsilosis are a recognized but less common cause of nail infection and that Candida nail infections tend to occur when predisposing conditions like immunosuppression and diabetes are present, or in chronic mucocutaneous candidiasis.^[19][10][20] Case reports and reviews of chronic mucocutaneous candidiasis describe patients with persistent Candida infections of skin, mucosa, and nails, sometimes including all toenails, in association with specific primary immune defects, showing that systemic immune problems can present with chronic Candida onychomycosis.^[2][8]

Contradicts

High-quality reviews and clinical overviews of onychomycosis consistently state that the common cause of toenail fungus is dermatophytes (especially Trichophyton rubrum), not Candida, and that yeasts account for only a minority (roughly 10–20%) of nail infections.^{[12][14][19][22]} These sources describe recurrent onychomycosis as multifactorial, with high recurrence rates attributed to factors such as incomplete eradication of the pathogen, reinfection from untreated tinea pedis, nail trauma, older age, diabetes, and other local or host factors, rather than systemic Candida overgrowth as the usual cause.^[17][18][19] Candida nail infections are described as typically occurring in specific settings (chronic mucocutaneous candidiasis, immunodepression, diabetes, frequent water exposure) and as one of several possible etiologies, not as the default explanation for recurrent toenail fungus.^{[10][13][19][20]} No guideline-level or review evidence supports the proposition that recurrent onychomycosis is "almost always" a sign of systemic Candida overgrowth; instead, Candida is a minority cause and systemic candidiasis is usually a serious invasive condition not diagnosed based on toenail findings alone.^[19] Likewise, mainstream medical literature does not identify environmental mold exposure as a common or established cause of immune suppression leading specifically to systemic Candida overgrowth and recurrent nail fungus; while severe immunosuppression from conditions like HIV or immunosuppressive drugs is well recognized, mold exposure is not cited in standard onychomycosis reviews as a typical driver.^[19][20]

Mainstream view

The mainstream medical view is that recurrent toenail fungus (onychomycosis) is most often caused by dermatophyte fungi, with yeasts such as Candida and nondermatophyte molds accounting for a smaller proportion of cases.^{[12][14][19][22]} Recurrent disease is common even after appropriate therapy and is attributed to factors such as persistent or reinfection by the same pathogen, coexisting tinea pedis, nail deformity, host comorbidities (age, diabetes, vascular disease), and sometimes defective immunity, but not routinely to systemic Candida overgrowth.^[17][18][19] Candida onychomycosis is recognized, particularly in patients with chronic mucocutaneous candidiasis or other forms of immunodepression, and Candida nail disease can be one manifestation of broader immune problems, but this is not considered the typical explanation for the majority of recurrent toenail infections in otherwise healthy individuals.^{[10][13][19][20]} Environmental mold exposure is not cited in major reviews or clinical overviews of onychomycosis as a usual cause of immune suppression leading to Candida overgrowth; mainstream practice focuses on confirming the specific pathogen by mycologic testing and addressing standard risk factors rather than assuming systemic Candida driven by mold exposure.^[19][20]

In their own wordsWatch sourceArchived copy

“Toenail fungus that keeps coming back is almost always a sign of systemic Candida overgrowth, which thrives when the immune system is suppressed by mold exposure.”

Rule: RSMo § 331.010(1)

See every doc bro who says they can treat or advise on Mold illness and CIRS

Jaban M Moore is not licensed or approved by Missouri State Board of Chiropractic Examiners to advertise Heel pain is the hallmark symptom of Bartonella, a bacterial co-infection commonly found alongside Lyme. as within their scope of practice.

Heel pain is the hallmark symptom of Bartonella, a bacterial co-infection commonly found alongside Lyme.

Supports

High-quality evidence confirms that Bartonella infection can cause musculoskeletal symptoms (myalgia, arthralgia, arthritis, tendinitis, osteomyelitis) and can present with rheumatologic complaints, including pain in bones and joints.^[21][25] Bartonella species are recognized in peer‑reviewed literature as significant co‑infections in Lyme disease patients, meaning Bartonella can commonly be found alongside Lyme infection.^[24][23] Clinical and case‑series data in rheumatologic cohorts from Lyme‑endemic regions show associations between Bartonella bacteremia and symptoms such as myalgia and joint pain, supporting that Bartonella can be a pain‑associated co‑infection in this context.^[20][25]

Contradicts

The peer‑reviewed literature does not identify heel pain, or plantar heel pain, as a hallmark or defining symptom of Bartonella infection. Descriptions of musculoskeletal manifestations of cat scratch disease (a Bartonella infection) list myalgia, arthropathy, tendinitis, neuralgia, and osteomyelitis, but do not single out heel pain or plantar enthesopathy as characteristic.^[21] In Lyme disease resources, heel‑specific pain is more often linked to Borrelia infection itself rather than Bartonella, suggesting heel pain is not uniquely or specifically tied to Bartonella.^[22] Existing Bartonella rheumatology studies from Lyme‑endemic regions acknowledge that they cannot establish a causal link between Bartonella infection and rheumatic symptoms such as myalgia and joint pain, indicating that even general pain associations are not firmly established, let alone a specific hallmark like heel pain.^[20][25] No major guidelines, systematic reviews, or randomized trials in the index list provided address Bartonella or identify heel pain as a hallmark symptom; the index papers concern hypertension, nutrition, headache, transfusion, or methods rather than Bartonella manifestations.

Mainstream view

Mainstream infectious‑disease and rheumatology literature recognizes Bartonella as a cause of cat scratch disease and other syndromes that can include musculoskeletal pain (myalgia, arthralgia, arthritis) and, in some contexts, rheumatologic presentations.^[21][25] Bartonella is accepted as a possible co‑infection in some Lyme disease patients, but the evidence base for specific symptom patterns is limited, and causal relationships between Bartonella bacteremia and chronic pain syndromes remain uncertain.^{[20][23][24][25]} Conventional medical sources do not describe heel pain or plantar heel pain as a hallmark or defining symptom of Bartonella; instead, heel pain is typically attributed to local musculoskeletal conditions such as plantar fasciitis, Achilles tendinopathy, or other biomechanical causes, and when heel pain occurs in Lyme disease it is usually discussed in relation to Borrelia rather than Bartonella.^[21][22] Therefore, the mainstream view is that Bartonella may contribute to nonspecific musculoskeletal pain as a co‑infection but heel pain is not considered a hallmark symptom of Bartonella in peer‑reviewed, guideline‑level evidence.

In their own wordsWatch sourceArchived copy

“Heel pain is the hallmark symptom of Bartonella, a bacterial co-infection commonly found alongside Lyme.”

Rule: RSMo § 331.010(1)

See every doc bro who says they can treat or advise on Lyme disease

Jaban M Moore is not licensed or approved by Missouri State Board of Chiropractic Examiners to diagnose, treat, or cure Numbness or tingling in the feet can indicate Lyme related small fiber neuropathy or leaky gut..

Numbness or tingling in the feet can indicate Lyme related small fiber neuropathy or leaky gut.

Supports

Lyme disease can cause peripheral nervous system involvement, and CDC guidance states that when peripheral nerves are affected, patients can develop radiculoneuropathy with numbness, tingling, shooting pain, or weakness in the arms or legs. ^[3][18][22] The indexed Lyme/PTLDS study reported that small fiber neuropathy appeared to be associated with post-treatment Lyme disease syndrome and may be responsible for certain sensory symptoms . ^{[5][19][20][21]} Another peer-reviewed report in the search results concluded that painful small fiber neuropathy may be a manifestation of Lyme disease, but this was based on a small conference abstract rather than high-level evidence. ^[1][6]

Contradicts

The claim is too broad because numbness or tingling in the feet is nonspecific and has many more common causes than Lyme disease, so the symptom alone does not indicate Lyme-related neuropathy. The evidence linking Lyme to small fiber neuropathy is limited, largely observational, and not yet established by high-quality systematic reviews or randomized trials. ^{[6][19][20][21]} The 'leaky gut' part is not supported by the indexed papers, and no major guideline in the provided set treats leaky gut as a validated explanation for distal numbness or tingling. ^[5] The only indexed nutrition/gastroenterology guidelines listed are about inflammatory bowel disease and parenteral nutrition, not leaky gut as a neuropathy diagnosis . ^[4][2][3]

Mainstream view

Mainstream medicine recognizes that Lyme disease can sometimes cause neurologic symptoms, including peripheral neuropathy with numbness or tingling, but diagnosis requires clinical evaluation and objective evidence rather than symptoms alone. ^[4][2] Small fiber neuropathy may occur in some patients with Lyme or post-treatment Lyme disease syndrome, but the evidence base is limited and not sufficient to say that foot tingling 'indicates' Lyme-related small fiber neuropathy. ^{[6][5][18][19][20][21]} 'Leaky gut' is not a mainstream evidence-based explanation for numbness or tingling in the feet. ^[1]

In their own wordsWatch sourceArchived copy

“Numbness or tingling in the feet can indicate Lyme related small fiber neuropathy or leaky gut.”

Rule: RSMo § 331.010(1)

See every doc bro who says they can treat or advise on Lyme disease

Jaban M Moore is not licensed or approved by Missouri State Board of Chiropractic Examiners to diagnose, treat, or cure Cracked heels can point to an underactive thyroid or diabetes because both conditions reduce circulation and the skin's ability to retain moisture..

Cracked heels can point to an underactive thyroid or diabetes because both conditions reduce circulation and the skin's ability to retain moisture.

Supports

The influencer’s specific mechanism (reduced circulation and impaired moisture retention) is oversimplified, but there is moderate evidence that both diabetes and hypothyroidism are associated with dry, fissured foot skin, so cracked heels can be one of many possible cutaneous signs rather than a reliable diagnostic marker. ^[6][24][25] Observational data in diabetic cohorts show high prevalence of xerosis and fissures on the feet, with dryness and cracking recognized as common skin manifestations of diabetic neuropathy and autonomic dysfunction, and as risk markers for later foot ulceration and infection. ^[26][27] Prospective and cross‑sectional studies of diabetic foot demonstrate that plantar xerosis, fissures and callus formation are frequent and clinically relevant findings in diabetes, supporting an association between diabetes and cracked heels as part of diabetic foot skin changes. Expert reviews and educational resources on diabetic foot care consistently describe very dry, peeling, cracking skin and heel fissures as typical xerosis-related lesions in people with diabetes, linked in part to neuropathy and sudomotor dysfunction that reduce sweat and oil production and thus impair the skin barrier. Academic and clinical reviews of xeroderma/xerosis list diabetes mellitus and hypothyroidism among systemic causes of abnormal dryness and cracking of the skin, implying that these endocrine disorders can contribute to xerosis that may present as cracked heels, although they are not described as a specific diagnostic sign. ^[2] Some dermatologic case series and reviews of palmoplantar keratoderma note that hypothyroidism is a rare but documented cause of acquired thickening and fissuring of palm/sole skin, suggesting that severe or unusual heel fissures can, in uncommon cases, be related to hypothyroid-associated keratoderma.

Contradicts

There is no high-quality evidence (e. ^[6] g. , randomized controlled trials, large prospective cohorts, or major guideline statements) indicating that cracked heels alone are a strong or specific clinical indicator that someone has an underactive thyroid or diabetes, and cracked heels are common in the general population from purely mechanical and environmental causes such as open-back shoes, prolonged standing, obesity, dry climate, and inadequate emollient use. ^[4][5] Reviews of heel fissures and xerosis emphasize multifactorial etiologies, with systemic diseases like diabetes and hypothyroidism listed as possible contributors but not singled out as primary or specific causes, and they highlight that anyone can develop cracked heels regardless of systemic disease status. Major endocrine and diabetes guidelines focus on established diagnostic criteria and complications (glycemic measures, neuropathy, macrovascular and microvascular disease, diabetic foot ulcers) and do not list cracked heels as a screening sign or diagnostic criterion for hypothyroidism or diabetes, indicating that the symptom is considered nonspecific and low-value for diagnosis compared with other clinical and laboratory findings. ^{[2][24][25][26]} Educational resources on diabetic feet stress that skin lesions such as xerosis, fissures, and calluses are common but emphasize neuropathy, peripheral arterial disease, and ulceration as clinically critical features, and they do not recommend using cracked heels in isolation to infer underlying systemic endocrine disease. ^[27] Overall, the literature treats cracked heels as a nonspecific manifestation of xerosis and mechanical stress, not as a focused marker that reliably “points to” hypothyroidism or diabetes in the way the influencer implies.

Mainstream view

The mainstream medical view is that cracked heels (heel fissures) are usually a nonspecific manifestation of xerosis and mechanical stress on the plantar skin, commonly influenced by footwear, standing, obesity, aging, climate, and skin-care practices. ^[24] Diabetes mellitus is widely recognized to increase the risk of foot xerosis and fissures through neuropathy and sudomotor dysfunction, and cracked, dry feet in a person with known diabetes are clinically important because they increase risk of infection and ulceration and warrant careful foot care and monitoring, but cracked heels are not used as a stand-alone diagnostic sign to identify diabetes. ^[25][26][27] Hypothyroidism is recognized to cause generalized dry, coarse skin and, more rarely, palmoplantar keratoderma and fissuring, so heel cracking can coexist with hypothyroidism as part of broader xerosis, yet it is not considered a specific or sensitive indicator of thyroid dysfunction, and clinicians rely on systemic symptoms and thyroid function tests for diagnosis. In standard practice, cracked heels are treated as a common, mostly benign skin condition that may occasionally reflect underlying systemic disease but, by itself, does not strongly “point to” diabetes or hypothyroidism; instead, persistent or severe heel fissures in combination with other systemic signs prompt further evaluation. Mainstream guidance therefore supports recognizing cracked heels as one possible, low-specificity cutaneous clue within a broader clinical picture, rather than as a direct diagnostic marker of underactive ^[2]

In their own wordsWatch sourceArchived copy

“Cracked heels can point to an underactive thyroid or diabetes because both conditions reduce circulation and the skin's ability to retain moisture.”

Rule: RSMo § 331.010(1)

See every doc bro who says they can treat or advise on Thyroid disease and Hashimoto

Jaban M Moore is not licensed or approved by Missouri State Board of Chiropractic Examiners to advertise Swollen ankles point to lymphatic congestion or kidney stress. as within their scope of practice.

Swollen ankles point to lymphatic congestion or kidney stress.

Supports

High-quality clinical references support that ankle swelling is a symptom of peripheral edema and can occur with renal disease, including chronic kidney disease and nephrotic syndrome, because renal fluid/sodium retention and protein loss can cause bilateral edema. ^[29][30][31] Major primary-care and specialty guidance also lists lymphatic disorders, including lymphedema, among recognized causes of chronic lower-extremity swelling. The presence of ankle swelling can therefore be consistent with kidney disease or lymphatic disease as part of a broad differential diagnosis. ^[28] The index papers provided do not directly test this claim; is unrelated to edema, and the remaining index items are not peer-reviewed evidence for this specific statement.

Contradicts

The claim is too specific and overinterprets a nonspecific sign. ^[28] Swollen ankles do not point uniquely to lymphatic congestion or kidney stress; they are more commonly approached as peripheral edema with a broad differential that includes heart failure, venous insufficiency, medications, liver disease, nephrotic syndrome, pregnancy, and other causes. ^[29][30][31] Clinical guidance emphasizes that bilateral lower-extremity edema is often systemic and requires evaluation rather than assuming lymphatic congestion or kidney stress. Evidence is weak for using ankle swelling alone as a diagnostic indicator of “lymphatic congestion,” because lymphedema usually has additional clinical features and is not the most common explanation for ankle swelling.

Mainstream view

Mainstream medicine views swollen ankles as a nonspecific sign of peripheral edema that can be caused by kidney disease, lymphedema, heart failure, venous disease, medications, liver disease, and other conditions. ^[29][31] Kidney disease and lymphatic disorders are on the differential, but ankle swelling by itself does not reliably indicate either one, and it should prompt clinical evaluation based on laterality, duration, associated symptoms, and exam findings. ^[28][30]

In their own wordsWatch sourceArchived copy

“Swollen ankles point to lymphatic congestion or kidney stress.”

Rule: RSMo § 331.010(1)

Jaban M Moore is not licensed or approved by Missouri State Board of Chiropractic Examiners to advertise Cold feet are one of the most overlooked signs of hypothyroidism. as within their scope of practice.

Cold feet are one of the most overlooked signs of hypothyroidism.

Supports

Cold intolerance and feeling unusually cold, including in the extremities, is a well-recognized symptom of hypothyroidism in reviews, textbooks, and guidelines. ^{[2][6][33][34][36]} Multiple authoritative clinical overviews list cold intolerance as a common symptom of hypothyroidism and describe decreased skin blood flow leading to cold skin, particularly in the hands and feet. ^[7] These sources explain that reduced thyroid hormone lowers basal metabolic rate and heat production and is associated with cutaneous vasoconstriction, so extremities may feel cold even in otherwise normal environments. Several educational and review-type sources explicitly note that people with hypothyroidism often report cold hands or feet and that cool extremities are a possible sign in hypothyroid patients. ^[5] This supports the narrower part of the claim that cold feet can occur as a symptom of hypothyroidism, and that, in a symptomatic hypothyroid patient, cold extremities may be one of several clinical clues pointing toward the diagnosis. ^[35]

Contradicts

High-quality evidence does not support the stronger framing that cold feet are one of the most overlooked signs of hypothyroidism. ^[6][34][36] Standard clinical reviews and guidelines emphasize nonspecific but common symptoms such as fatigue, weight gain, dry skin, constipation, and cold intolerance in general, but they do not single out cold feet specifically as a key or especially overlooked sign. ^[2][35] Epidemiologic work on cold extremities in the general population shows that cold hands and feet are common, especially in women, and are associated with multiple other symptoms and behavioral factors rather than specifically with thyroid disease; this suggests cold feet are a nonspecific complaint with many potential causes. Some clinical sources also stress that classic features like cold intolerance may be absent altogether in many patients with hypothyroidism, indicating that reliance on any single symptom, including cold feet, is unreliable. ^[33] Overall, no systematic reviews, meta-analyses, randomized trials, or major practice guidelines were found that quantify cold feet as a particularly sensitive, specific, or commonly missed sign for hypothyroidism, so the claim that it is “one of the most overlooked signs” is not evidence-based and appears exaggerated. ^[1]

Mainstream view

The mainstream medical view is that hypothyroidism commonly presents with nonspecific symptoms such as fatigue, weight gain, constipation, dry skin, and particularly generalized cold intolerance, and that some patients may also have cold or cool extremities as part of this picture. ^[34][36] However, cold feet alone are considered a nonspecific symptom with many other possible explanations (e. g. , normal variation, peripheral vascular issues, autonomic or functional disorders, environmental exposure), so they are not regarded as a distinctive or prioritized sign of hypothyroidism on their own. Current guidelines and high-quality reviews recommend diagnosing hypothyroidism based on serum TSH and thyroid hormone levels in the context of the overall clinical picture, not on isolated symptoms like cold feet, and they do not describe cold feet as a commonly overlooked hallmark sign. ^[2][6][35]

In their own wordsWatch sourceArchived copy

“Cold feet are one of the most overlooked signs of hypothyroidism.”

Rule: RSMo § 331.010(1)

See every doc bro who says they can treat or advise on Thyroid disease and Hashimoto